I-cardiomyopathy ye-hypertrophic

Lesi sifo, esiqinisa udonga lwesobunxele, futhi ezimweni ezingavamile kakhulu ze-ventricle yenhliziyo efanele, kuthiwa i-hypertrophic cardiomyopathy (HCMC). Kulesi sifo, ukuqina kwamacala amabi kakhulu kunamabonakude, ngakho-ke i-septum ye-interventricular ivame ukulimala.

Kukholelwa ukuthi lesi yisifo sabathameli - kungenxa yokuzikhandla ngokomzimba ukuthi i-hypertrophy ivela. Sisazi amacala ambalwa lapho abagibeli befa ngenxa yezemidlalo ngenxa ye-hypertrophic cardiomyopathy - umdlali we-Hungarian we-football Miklos Feher nomdlali wezemidlalo waseMelika uJesse Marunde.

Kulesi sifo, imicu ye-muscle ku-myocardium inezindawo eziyingozi, ehambisana nokushintshashintsha kwezakhi zofuzo.

Amafomu we-hypertrophic cardiomyopathy

Namuhla, odokotela bachaza izinhlobo ezintathu ze-hypertrophic cardiomyopathy:

  1. Isivimbelo esisisekelo - i-gradient ekuphumuleni inkulu kunaleyo noma ilingana no-30 mm Hg. Art.
  2. Ukuvimbela ukubhubhisa - ukuguquguquka okuzenzakalelayo kwe-gradient yangaphakathi.
  3. Ukuvinjelwa okuvame ukuhamba kwesikhathi - isimo sokuthula esingaphansi kuka-30 mm Hg. Art.

Ukuvimbela i-hypertrophic cardiomyopathy kufana nalezi zinhlobo ezintathu zesifo, kanti ifomu elingavinjelwe ngempela libhekene nesimo se-stenosis esingaphansi kuka-30 mm Hg. Art. esimweni esinokuthula nesicasulayo.

Izimpawu ze-hypertrophic cardiomyopathy

Ukubonakaliswa kwe-hypertrophic cardiomyopathy kungase kungabi khona - iziguli ezingaba ngu-30 azikwenzi izikhalazo, lapho kwenzeka ukufa okungazelelwe kungaba ukuphela kwesibonakaliso saleso sifo. Ezindaweni ezikhethekile zobungozi kukhona iziguli ezincane ezingaboni izikhalazo, ngaphandle kokuphazamiseka kwenhliziyo.

Ngoba lesi sifo sibonakala yi-okuthiwa i-small-emission syndrome - kulokhu, ukuphelelwa yisikhathi kwenzeka, ukuphefumula nokuphefumula, nokuhlaselwa kwe- angina kwenzeka.

Futhi, nge-cardiomyopathy ye-hypertrophic, kungase kube khona ukubonakaliswa kwesokunxele senhliziyo ye-ventricular, engase ikhule ibe yincwadzi ye-congestive ye-heart.

Ukwehluleka ngesigqi senhliziyo kungabangela ukuphelelwa amandla . Ngokuvamile lezi ziyi-extrasystoles ze-ventricular kanye nama-paroxysms we-tricycardia ye-ventricular.

Ezimweni ezingavamile kakhulu, iziguli zingase zibe ne-endocarditis ezithathelwanayo kanye ne-thromboembolism.

Ukuxilongwa kwe-hypertrophic cardiomyopathy

Ngokungafani nezinye izinhlobo ze-cardiomyopathy, ifomu le-hypertrophic litholakala ukuthi lilula kakhulu ngenxa yesimiso esinqunyiwe: ukuze ukuxilongwa kutholakale, ukuqina kwe-myocardial kufanele kube mkhulu kunalokho noma kulingane no-1.5 cm kanye nokuba khona kokungasebenzi kahle kwe-ventricular (ukuphumula okungafanelekile).

Uma kuhlolwe, isiguli sitholakale ukwandisa umngcele wenhliziyo ngakwesobunxele, futhi uma uvinjelwe, umsindo uzwakala (i-systolic rhomboid).

Phakathi kwezindlela ezengeziwe zokutadisha lesi sifo kukhona okulandelayo:

Ukwelashwa kwe-cardiomyopathy ye-hypertrophic

Ukuxilongwa nokuphathwa kwe-cardiomyopathy ye-hypertrophic kuyahambisana kakhulu ukuvimbela imiphumela ebulalayo. Ngemva kokuhlola ukuchazwa kwenkinga yesifo, uma kungenzeka ukuthi umphumela obulalayo, ukwelashwa okuyinkimbinkimbi kwenziwa. Uma kungekho usongo lokufa, futhi izimpawu azikho ziboniswa, kanti ukwelashwa okukhethekile akukwenziwa.

Ukwelashwa kubaluleke kakhulu ukunciphisa umsebenzi womzimba, futhi nokuthatha izidakamizwa nge-ionotropic effect engafanele. Lesi sigaba sihlanganisa abakwa-beta-blockers kanye nabaphikisi be-calcium. Bazikhethela ngabanye, futhi kunikezwa ukuthi ukwamukela kwenziwa isikhathi eside (kuze kube yilapho kuthola khona impilo yonke), namuhla odokotela bazama ukunikeza imithi eneemiphumela emibi kakhulu. Kwakusetshenziswe u-Anaprilin ngaphambili, futhi namuhla kunezinhlobo eziningi zezizukulwane ezintsha.

Futhi, imishanguzo ye-antiarrhythmic kanye nama-antibiotic asetshenziselwa ukwelashwa uma kwenzeka isifo esithathelwanayo se-pathology.